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Glucocorticoid-transactivated TSC22D3 attenuates hypoxia- and diabetes-induced Müller glial galectin-1 expression via HIF-1α destabilization.

Galectin-1/LGALS1, a newly acknowledged angiogenic issue, contributes to the pathogenesis of diabetic retinopathy (DR).Galectin-1/LGALS1, a newly acknowledged angiogenic issue, contributes to the pathogenesis of diabetic retinopathy (DR).

Not too long ago, we demonstrated that glucocorticoids suppressed an interleukin-1β-driven inflammatory pathway for galectin-1 expression in vitro and in vivo. Right here, we present glucocorticoid-mediated inhibitory mechanism in opposition

Compensatory Hippocampal Neurogenesis within the Absence of Cognitive Impairment Following Experimental Hippocampectomy in Grownup Rats

Compensatory Hippocampal Neurogenesis in the Absence of Cognitive Impairment Following Experimental Hippocampectomy in Adult RatsCompensatory Hippocampal Neurogenesis in the Absence of Cognitive Impairment Following Experimental Hippocampectomy in Adult Rats

Temporal lobe epilepsy (TLE) is the most common kind of focal epilepsy in grownup people, and hippocampal sclerosis (HS) is the principle pathological discovering in any such epilepsy. In refractory